Anya Medina Benavente, post-doc
My name is Anya Medina. I received my Medical Degree from Catolica Santa Maria University in Peru in 2007. During my medical studies and my first year working as a general medical doctor, I developed a great interest in understanding basic science to explain diseases. Therefore, I decided to join a Cell Physiology Ph.D. program at Gunma University in Japan. My Ph.D. studies were focused on Beta cell physiology and regeneration. Throughout my Ph.D. in Japan I developed numerous new technical and intellectual skills and a deep understanding and interest in beta cell biology. The translation of this basic science knowledge into therapy for important diseases like diabetes encouraged me to pursue an academic career as a scientist.
Upon completion of my PhD I was fortunate to be recruited to join the Lund University Diabetes Center and I joined the Unit of Molecular Metabolism in 2015. During my first 3 years of postdoctoral experience, I have been able to fully characterize the autophagy pathways in beta cells and study how these processes are related to beta cell dysfunction in diabetes.
Retrieved from Lund University's publications database
- Differential effects of three echovirus strains on cell lysis and insulin secretion in beta cell derived lines.
- Glycogen metabolism in the glucose-sensing and supply-driven β-cell
- Serotonin (5-HT) and 5-HT2A receptor agonists suppress lipolysis in primary rat adipose cells
- Serotonin (5-HT) receptor 2b activation augments glucose-stimulated insulin secretion in human and mouse islets of Langerhans.